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Type of alopecia hair loss


Alopecia is premature hair fall or absence of one or more body parts. Its causes are many and the types of alopecia are classified as follows:


 Non-scarring alopecia:
They are very common and can be improved or cured through treatment and even some revert spontaneously without being treated. In this type of alopecia the hair follicle has a pathological behavior but still alive except alopecia extending over long periods (common or androgenetic alopecia).


Androgenic Alopecia:
 It is also known as androgenetic alopecia, or pattern baldness premature. It affects many men and women rarely.

 
If you suffer from androgenetic alopecia, like most of the men I recommend you take a look to the left and check the Norwood Scale what type / degree of alopecia have.
You will be very useful to know if a treatment can give results. If your degree is very advanced (5, 6 or 7) have to think about surgery or just accept that you are bald. There are no treatments that have proven effective in such advanced degrees.



Androgens are the hormones responsible for hair loss and that is why many current treatments inhibit the production thereof.
If progress continues baldness and hair follicles die, the process becomes irreversible and no treatment exists to restore the situation. It can only be repaired by transplanting living in other areas towards the crown follicles and entries; microfollicular capillary. Ideally, of course, is to fight the fall while the follicles are alive.
Women are luckier than men because androgenetic alopecia appears only on the front of the scalp, leaving hair thinning but rarely gets to have completely depopulated areas and in man.
 


 Alopecia areata: the cause is unknown, but its symptoms are clear: round patches on the head of hair completely depopulated. Alopecia totalis (AT) is when the scalp hair is lost completely. In cases of Alopecia universalis (AU), the whole body hair is lost.

 
There are no fully effective treatments for this type of alopecia yet. The therapies used to average results are: ultraviolet light, topical corticosteroids, steroid injections or irritating to excite the follicles hair growth stimulating agents.


Traumatic alopecia: can be caused by the use of hair dryers, metal combs or any other element capable of generating lesions on the scalp. Can also be generated when the patient-in a manic state to be up in himself. This condition is known as trichotillomania.

 Diffuse alopecia (chronic telogen effluvium): el term "telogen effluvium" coined by Kligman fue en el año 1961. Es la del pérdida by acute systemic behind enfermedades chronic, emotional stress, febrile enfermedades delivery. En latter, telogen effluvium el puede last six months hasta luego to fully recuperarse.

 Alopecia drugs the drugs: vitamin A la dosis en large, these cytostatics, Antithyroid Agents, anticoagulates, el Mercurio y el valproic acid capaces son of producir alopecia. Cuando la la alopecia drug is suspended disappears.

Alopecia systemic enfermedades: origen endocrine, infectious, Lupus erythematosus nutritional deficit.

Alopecia hereditary syndromes: case en el de la congenital atriquia, el individio lacks the. También la met en la temporal triangular alopecia, en el de la pérdida del cabello en anagen syndrome, hypoplasia en la y el del by cartilage, en el Menkes syndrome, anhidrótica en la y el en tricorrinofalángico ectodermal dysplasia syndrome.



 Scarring alopecia: 

 Unfortunately, this type of alopecia usually irreversible because there is damage, malformation or complete rupture of the follicular structure. There is no treatment or drug that helps in hair follicles inert. If there are no damaged areas can be appealed to a hair transplant. Scarring alopecia are classified as follows:
 
infectious alopecia, fungi (Kerion, candidiasis, favus); Bacteria (syphilis, leprosy, skin debris); Virus (herpes, chickenpox); Protozoal infections. (Leishmaniasis)

 Alopecia by physicochemical agents, caustic burns and mechanical injury. rediodermitis X-ray determined that the follicles are more sensitive to radiation.

 Tumor alopecia: y dermal tumor metastasis. Mast cells, the basal squamous cell carcinomas, lymphomas y anexiales tumors.

  Alopecia by dermatosis: Graham-Little syndrome, dermatomyositis, sarcoidosis and follicular mucinosis.

 Alopecia for hereditary diseases: porokeratosis of Mibelli, epidermal nevus, Darier's disease, ichthyosis and aplasia cutis.

 Alopecia clinical syndromes decalvantes: Erosive pustular dermatosis, decalvans folliculitis, alopecia and parvimaculata pseudopelade.

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